During this time, loved ones confront the alcoholic about the problem and the negative ways in which addiction is impacting his or her life. An intervention involves careful planning, acquiring of relevant knowledge, development sober house of consequences each loved one might enact if treatment is denied, and follow-up with the sufferer to ensure a successful recovery process. This shortness of breath, however, will also often occur simply upon lying down.
While relatively rare, alcohol becomes more likely to cause cardiomyopathy with excessive alcohol use. Excessive alcohol use is defined by the CDC as more than 8 drinks per week for women and more than 15 drinks per week for men. Alcohol-induced cardiomyopathy, especially when more severe, leads to deadly problems like heart attack, stroke or heart failure. Individuals with this condition who don’t stop drinking heavily are at the greatest risk.
Types of Cardiomyopathy
The use of carvedilol, trimetazidine with other conventional heart failure drugs have been proven to be beneficial in some studies. Acute can be defined as large volume acute consumption of alcohol promotes myocardial inflammation leading to increased troponin concentration in serum, tachyarrhythmias including atrial fibrillation and rarely ventricular fibrillation. Most common age population for ACM is males from age with significant history of alcohol use for more than 10 years. Females constitute roughly 14 % of cases of alcohol induced cardiomyopathy however lifetime exposure required for women to develop alcohol induced cardiomyopathy is less compared to men.
Renaud and de Lorgeril  suggested that the inhibition of platelet reactivity by wine may be one explanation for protection from CAD in France. Results from serum chemistry evaluations have not been shown to be useful for distinguishing patients with alcoholic cardiomyopathy (AC) from those with other forms of dilated cardiomyopathy (DC). However, results from tissue assays have been shown to be potentially helpful in distinguishing AC from other forms of DC. In addition, people who receive early treatment for ACM, including medication and lifestyle modifications, have a better chance of improving their heart function and overall health.
When someone has alcoholic cardiomyopathy, they have a form of cardiomyopathy caused by alcoholism. Alcoholic cardiomyopathy is most often diagnosed in older adults, with an average age of onset being 50 years old. In addition to adding to the heart’s workload, Brown says, extra pounds can make you sluggish and hinder physical activity.
Experimental studies analysing the depressive properties of alcohol on the cardiac muscle invariably use similar approaches[31-39]. Accordingly, a given amount of alcohol is administered to volunteers or alcoholics, followed by the measurement of a number of haemodynamic parameters and, in some cases, echocardiographic parameters. Generally, following alcohol intake, healthy, non-drinking individuals showed an increase in cardiac output due to a decline in peripheral arterial resistance and an increase in cardiac frequency. However, during the time that these haemodynamic changes appeared, some researchers identified a possible decrease in the ejection fraction and other parameters related to systolic function[32-39]. This was questioned by other authors, who pointed out that these conclusions could not be drawn, as alcohol itself also induces changes in the pre-load and after-load conditions, which influence cardiac contractility. However, in this context, experimental in vitro studies using cardiomyocytes have shown that alcohol depresses the contractile capacity of the myocardium, regardless of the sympathetic tone and the haemodynamic conditions.